The Serine/Threonine Protein Kinase ULK1 pipeline drugs market research report outlays comprehensive information on the Serine/Threonine Protein Kinase ULK1 targeted therapeutics, complete with analysis by indications, stage of development, mechanism of action (MoA), route of administration (RoA), and molecule type. GlobalData’s report assesses the drugs in the Serine/Threonine Protein Kinase ULK1 pipeline by therapy areas, indications, stages, MoA, RoA, molecule type and the key players in the development pipeline. Buy the report here.
The report also covers products from therapy areas such as Oncology which include the indications Solid Tumor, and Oncology. It also reviews key players involved in Serine/Threonine Protein Kinase ULK1 targeted therapeutics development with respective active and dormant or discontinued products.
The Serine/Threonine Protein Kinase ULK1 pipeline targets constitutes close to six molecules. Out of which, approximately four molecules are developed by companies and the remaining by the universities/institutes. The molecules developed by companies in Phase II, Preclinical, and Discovery stages are 1, 2, and 1 respectively. Similarly, the universities portfolio in Preclinical, and Discovery comprises 1, and 1 molecule.
Serine/Threonine Protein Kinase ULK1 overview
Forkhead Box Protein P3 (ULK1) is a serine/threonine protein kinase that plays a central role in the initiation of autophagy. ULK1 is a key regulator of the autophagy machinery and is essential for maintaining cellular homeostasis, responding to stress, and eliminating damaged or unnecessary cellular components. ULK1 is part of the ULK kinase complex, which also includes ULK2, Atg13, and FIP200. This complex is activated in response to various cellular stresses, such as nutrient deprivation, oxidative stress, or the presence of damaged organelles. Upon activation, ULK1 phosphorylates downstream targets, initiating the formation of the autophagosome, a double-membraned vesicle that engulfs cellular material for subsequent degradation. Autophagy mediated by ULK1 is critical for cellular survival and adaptation to changing environmental conditions. Dysregulation of autophagy has been implicated in various diseases, including neurodegenerative disorders, cancer, and metabolic diseases.
For a complete picture of Serine/Threonine Protein Kinase ULK1’s drug pipeline, buy the report here.
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