The Mineralocorticoid Receptor pipeline drugs market research report outlays comprehensive information on the Mineralocorticoid Receptor targeted therapeutics, complete with analysis by indications, stage of development, mechanism of action (MoA), route of administration (RoA), and molecule type. GlobalData’s report assesses the drugs in the Mineralocorticoid Receptor pipeline by therapy areas, indications, stages, MoA, RoA, molecule type and the key players in the development pipeline. Buy the report here.
The report also covers products from therapy areas such as Cardiovascular, Genito Urinary System, Ophthalmology, and Gastrointestinal which include the indications Congestive Heart Failure (Heart Failure), Diabetic Hypertension, Chronic Kidney Disease (Chronic Renal Failure), Diabetic Nephropathy, Diabetic Retinopathy, Dry (Atrophic) Macular Degeneration, Liver Failure (Hepatic Insufficiency), and Metabolic Dysfunction-Associated Steatohepatitis (MASH). It also reviews key players involved in Mineralocorticoid Receptor targeted therapeutics development with respective active and dormant or discontinued products.
The Mineralocorticoid Receptor pipeline targets constitutes close to nine molecules. Out of which, approximately nine molecules are developed by companies and the remaining by the universities/institutes. The molecules developed by companies in Phase III, Phase II, Phase I, and Preclinical stages are 3, 2, 1, and 2 respectively.
Mineralocorticoid Receptor overview
Mineralocorticoid receptor (NR3C2) is a protein that in humans is encoded by the NR3C2 gene. It is a receptor both for mineralocorticoids (MC) such as aldosterone and for glucocorticoids (GC) such as corticosterone or cortisol. It binds to mineralocorticoid response elements (MRE) and transactivates target genes. The effect of MC is to increase ion and water transport and thus raise extracellular fluid volume and blood pressure and lower potassium levels.
For a complete picture of Mineralocorticoid Receptor’s drug pipeline, buy the report here.
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